Cellular Metabolism Case Study Part 1: 1. Given the autopsy report the massive cell nail in the kidneys, lungs, thymus, and heart indicate that the damage occurred at the cellular level, the parts of cellular respiration that occur in the mitochondrion. more(prenominal) than specifically, the poison directly affected the mitochondrion and in doing so it suggests that cellular respiration was disrupted. 2. The cellular deal that would be impaired by arsenate would affect the cellular wait on of glycolysis which occurs in the cytosol of a cell. In the information provided in the case study it states that arsenate glow replace inorganic phosphate in the timber of glycolysis that produces 1, 3-biphospholglycerate from glyceraldehyde 3-phosphate. This yields 1-arseno-3-phosphoglycerate. Therefore, glycolysis proceeds but the ATP molecule that would be generated from 1, 3-biophosphoglycerate is lost. 3. The cellular processes that would be impaired by rot enone would affect the cellular process of the negatron go chain which occurs within the mitochondrion of a cell. In information provided it states that rotenone is an agent that would inhibit the electron screw chain within the mitochondrial therefore would affect the electron embark chain. Part 2: 1. I think that the specific cellular process that was affected by the poison was the first tone of voice in the electron transport chain. The very low levels of nicotinamide ampere dinucleotide+ and the high levels of NADH indicate that the initial step in the electron transport chain was disrupted. Particularly, the function of NADH dehydrogenase was inhibited by the rotenone poison. NADH dehydrogenase is tame for converting the electron carrier, NADH, to NAD+. 2. If NAD+ had been administered to the women, she probably could have not been saved. The low levels of NAD+ in the cells are simply a symptom of the poison. The problem is that the rotenone prevents the transfer of electrons from NADH to the elect! ron transport...If you want to convey a full essay, order it on our website: OrderCustomPaper.com
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